Acute vs Chronic Gastritis | Chronic Gastritis vs Mob plab Gastritis Ua rau, Cov tsos mob, Kev kuaj mob thiab kev tswj hwm
Gastritis yog qhov mob ntawm lub plab mucosa. Nws yog ib qho kev kuaj mob histological, txawm hais tias qee zaum nws tau lees paub ntawm lub plab hnyuv sab sauv sab sauv (UGIE). Raws li qhov pib ntawm cov txheej txheem kab mob, nws yog categorized li mob plab thiab mob ntev. Kab lus no qhia txog qhov sib txawv ntawm qhov mob plab thiab mob ntev txog lub ntsiab lus, kev sib raug zoo ntawm lub cev, etiology, macroscopic thiab microscopic hloov pauv, kho cov yam ntxwv, teeb meem thiab kev tswj xyuas.
Acute Gastritis
Nws yog qhov mob mob ntawm lub plab mucosa, uas feem ntau erosive thiab hemorrhagic. Feem ntau ua rau muaj kev cuam tshuam yog kev siv cov tshuaj uas tsis yog-steroidal anti-inflammatory (NSAIDs), corticosteroids, raug rau cov tshuaj luminal ncaj qha xws li cawv, kev ntxhov siab xws li kub hnyiab, myocardial infarction, thiab intra cranial lesions thiab thaum lub sij hawm postoperative, chemotherapy thiab ischemia.
Endoscopically nws yog tus cwj pwm los ntawm diffuse hyperemia ntawm mucosa nrog ntau yam, me me, sab saum toj erosions thiab rwj. Microscopy nthuav tawm qhov kev raug mob ntawm qhov chaw epithelial thiab denudation thiab sib txawv necrosis ntawm cov qog nqaij hlav. Hemorrhage rau hauv lamina propria tuaj yeem pom. Cov hlwb mob tsis muaj nyob hauv ntau, txawm li cas los xij, cov neutrophils yog qhov tseem ceeb.
Hauv cov mob me, cov neeg mob feem ntau yog asymptomatic lossis tej zaum yuav muaj cov tsos mob me me. Hauv nruab nrab mus rau qhov hnyav, tus neeg mob muaj qhov mob epigastric, xeev siab, ntuav, haematemesis thiab melena. Hauv qhov xwm txheej hnyav, tus neeg mob tuaj yeem tsim qhov mob tob tob thiab perforations raws li qhov teeb meem.
Kev tswj tus mob plab feem ntau yog coj mus rau lub hauv paus. Lub sij hawm luv luv symptomatic therapy nrog antacids thiab acid suppression nrog proton twj tso kua mis inhibitors los yog antiemetics tej zaum yuav tsim nyog.
mob plab kab mob
Nws tau txhais histologically raws li kev nce ntawm cov lymphocytes thiab ntshav plasma hlwb hauv plab mucosa. Raws li etiology nws tau categorized li hom A, uas yog autoimmune hauv keeb kwm, hom B yog tshwm sim los ntawm Helicobacter pylori kab mob, thiab muaj ob peb ua rau tsis muaj hom uas nws etiology tsis paub.
Endoscopically, mucosa yuav tshwm sim atrophied. Microscopy qhia cov lympho-plasmatic infiltrate nyob rau hauv lub mucosa nyob ib ncig ntawm lub parietal hlwb. Neutrophils tsis tshua muaj. Mucosa tuaj yeem pom kev hloov pauv ntawm plab hnyuv metaplasia. Thaum kawg, mucosa yog atrophied nrog tsis tuaj parietal hlwb. Hauv kab mob H. Pylori, cov kab mob yuav raug sau tseg.
Feem ntau cov neeg mob gastritis ntev yog asymptomatic. Qee tus neeg mob tuaj yeem ua rau mob me me, tsis xis nyob, mob, xeev siab thiab anorexia. Hauv kev kuaj mob endoscopic, tej zaum yuav tsis muaj cov yam ntxwv lossis poob ntawm cov ntaub pua tsev ib txwm raug sau tseg. Txij li cov neeg mob no muaj kev pheej hmoo siab ntawm gastric carcinoma, kev tshuaj ntsuam endoscopically yuav tsim nyog. Cov neeg mob uas muaj hom A gastritis tuaj yeem muaj pov thawj ntawm lwm yam kabmob autoimmunity tshwj xeeb tshaj yog cov thyroid kab mob.
Raws li feem ntau ntawm cov neeg mob asymptomatic, lawv tsis xav tau kev kho mob. Cov neeg mob dyspepsia yuav tau txais txiaj ntsig los ntawm H. pylori eradication.
Dab tsi yog qhov txawv ntawm mob plab thiab mob plab ntev?
• Mob plab feem ntau yog erosive thiab hemorrhagic tab sis mob gastritis tsis yog.
• NSADs thiab cawv yog qhov ua rau mob plab hnyuv loj thaum autoimmunity thiab H Pylori yog qhov ua rau mob plab ntev.
• Endoscopically inflammatory pauv tau pom tsuas yog nyob rau hauv mob gastritis.
• Neutrophils yog cov kab mob loj tshaj plaws hauv kev mob plab mob plab thaum lympho-plasmatic infiltration pom nyob rau hauv mob gastritis.
• Cov kab mob gastritis muaj kev pheej hmoo ntawm plab hnyuv loj, tshwj xeeb tshaj yog hom A, uas suav tias yog ua ntej malignant.
